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PLoS pathogens
SARS-CoV-2 evolution balances conflicting roles of N protein phosphorylation.
A cohort-based study of host gene expression: tumor suppressor and innate immune/inflammatory pathways associated with the HIV reservoir size.
A single inactivating amino acid change in the SARS-CoV-2 NSP3 Mac1 domain attenuates viral replication in vivo.
Nuclear accumulation of host transcripts during Zika Virus Infection.
SIRT5 is a proviral factor that interacts with SARS-CoV-2 Nsp14 protein.
Distinct phenotype and function of circulating Vd1+ and Vd2+ ?dT-cells in acute and chronic hepatitis B.
Increased mucosal neutrophil survival is associated with altered microbiota in HIV infection.
Gut and blood differ in constitutive blocks to HIV transcription, suggesting tissue-specific differences in the mechanisms that govern HIV latency.
A20 upregulation during treated HIV disease is associated with intestinal epithelial cell recovery and function.
Increased HIV-1 transcriptional activity and infectious burden in peripheral blood and gut-associated CD4+ T cells expressing CD30.
